![Dab tsi yog Pathophysiology ntawm Ntshav Qab Zib Hom 1? Dab tsi yog Pathophysiology ntawm Ntshav Qab Zib Hom 1?](https://i.answers-medical.com/preview/medical-health/13835576-what-is-the-pathophysiology-of-diabetes-type-1-j.webp)
Video: Dab tsi yog Pathophysiology ntawm Ntshav Qab Zib Hom 1?
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2024 Tus sau: Michael Samuels | [email protected]. Kawg hloov kho: 2023-12-16 01:44
Hom 1 mob ntshav qab zib tshwm sim thaum qee qhov lossis tag nrho cov hlwb tsim cov tshuaj insulin hauv cov txiav txiav. Qhov no txhais tau tias nws pib thaum lub cev tiv thaiv kab mob tawm tsam cov cell hauv lub cev. Hauv hom 1 mob ntshav qab zib , lub cev tiv thaiv kab mob tsim cov tshuaj insulin (beta cells) hauv cov txiav txiav.
Ntxiv mus, dab tsi yog pathophysiology ntawm ntshav qab zib?
Cov pathophysiology ntawm ntshav qab zib cuam tshuam nrog qib insulin hauv lub cev, thiab lub cev muaj peev xwm siv cov tshuaj insulin. Feem ntau, pancreatic beta cells tso cov tshuaj insulin vim nce cov ntshav qabzib ntau ntau. Lub paj hlwb txhawm rau ua haujlwm ib txwm muaj tshwm sim tas li yuav tsum muaj cov piam thaj.
Ntxiv mus, yam xwm txheej ntawm hom 1 mob ntshav qab zib yog dab tsi? Hom 1 mob ntshav qab zib (tseem hu ua mob ntshav qab zib mob ntshav qab zib mellitus) yog kab mob autoimmune uas lub cev tiv thaiv kab mob tawm tsam thiab ua kom cov tshuaj insulin tsim tawm ntawm cov txiav txiav. Qhov poob ntawm cov insulin ua rau tsis muaj peev xwm tswj tau cov ntshav qab zib.
Dab tsi yog pathophysiology ntawm ntshav qab zib ketoacidosis?
DKA yog lub xeev ntawm qhov tsis txaus ntseeg lossis cov txheeb ze insulin tsis txaus los ntawm kev ua rau hyperglycemia, lub cev qhuav dej, thiab acidosis-ua rau derangements hauv cov metabolism nruab nrab. Qhov ua rau tshwm sim feem ntau yog pib mob, cuam tshuam kev kho tshuaj insulin, thiab pib tshiab mob ntshav qab zib . (Saib Etiology .)
Dab tsi yog Pathophysiology ntawm Ntshav Qab Zib Hom 2?
Cov pathophysiology ntawm hom 2 mob ntshav qab zib mob ntshav qab zib mellitus yog tus yam ntxwv ntawm cov tshuaj insulin tsis txaus, ua tsis tau zoo ntawm kev tsim cov piam thaj hauv lub cev, thiab tsis ua haujlwm cell -cell, thaum kawg ua rau -cell tsis ua haujlwm.
Pom zoo:
Yuav muaj dab tsi tshwm sim thaum ib tus neeg uas muaj ntshav qab zib hom 2 noj qab zib?
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Koj lub cell vam khom cov piam thaj rau lub zog. Hyperglycemia yog tus yam ntxwv ntawm ntshav qab zib - thaum cov ntshav qabzib ntau dhau vim tias lub cev siv tsis raug lossis tsis ua cov tshuaj insulin. Kev noj zaub mov ntau dhau yuav ua rau koj cov ntshav qab zib nce. Koj tau txais cov piam thaj los ntawm cov zaub mov koj noj
Cov ntshav qab zib li qub rau cov neeg laus uas tsis muaj ntshav qab zib yog dab tsi?
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Rau cov neeg feem coob uas tsis muaj ntshav qab zib, cov ntshav qab zib ua ntej noj mov nyob ib puag ncig 70 txog 80 mg/dL. Rau qee tus neeg, 60 yog qhov qub; rau lwm tus, 90 yog tus qauv
Dab tsi yog hom 2 mob ntshav qab zib mellitus nrog mob ntshav qab zib polyneuropathy?
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Mob ntshav qab zib neuropathy yog ib qho teeb meem loj thiab tshwm sim ntawm hom 1 thiab hom 2 mob ntshav qab zib. Nws yog hom kab mob puas tsuaj los ntawm cov ntshav qab zib hauv lub sijhawm ntev. Yog tias koj muaj ntshav qab zib thiab pom loog, tingling, mob, lossis tsis muaj zog ntawm koj txhais tes lossis taw, koj yuav tsum mus ntsib koj tus kws kho mob
Dab tsi ntawm cov hauv qab no yog qhov kev txhawj xeeb tseem ceeb ntawm cov kab mob ntshav qab zib?
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Ntawm qhov kev txhawj xeeb tseem ceeb yog tus kab mob tib neeg tiv thaiv kab mob (HIV) thiab kab mob siab B thiab kab mob siab C. Tus txheej txheem teeb tsa kev xav tau rau cov tswv ntiav haujlwm nrog cov neeg ua haujlwm raug cov ntshav lossis lwm yam khoom muaj peev xwm kis tau
Cov tshuaj hormones twg yog lub luag haujlwm rau ntshav qab zib mellitus thiab ntshav qab zib insipidus?
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Ua. Ob hom ntshav qab zib insipidus txuas nrog cov tshuaj hu ua vasopressin tab sis tshwm sim hauv ntau txoj kev. Vasopressin txhawb nqa cov dej nyob hauv ob lub raum