![Vim li cas hyperglycemia ua rau hyponatremia? Vim li cas hyperglycemia ua rau hyponatremia?](https://i.answers-medical.com/preview/medical-health/13996218-why-does-hyperglycemia-cause-hyponatremia-j.webp)
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2024 Tus sau: Michael Samuels | [email protected]. Kawg hloov kho: 2023-12-16 01:44
Sodium Kho rau Hyperglycemia . Xam qhov tseeb qib sodium hauv cov neeg mob nrog hyperglycemia . Hyperglycemia ua rau osmotic hloov dej los ntawm intracellular mus rau qhov chaw sab nrauv, ua ib tug txheeb ze dilutional hyponatremia.
Los ntawm qhov ntawd, hyperglycemia cuam tshuam rau qib sodium li cas?
Feem ntau piv txwv yog serum hyperglycemia . Kev sib sau ntawm cov piam thaj extracellular induces kev hloov ntawm dej dawb los ntawm qhov chaw intracellular mus rau qhov chaw extracellular. Ntshav sodium concentration yog diluted los ntawm qhov tseem ceeb ntawm 1.6 mEq/L rau txhua 100 mg/dL nce siab dua qub cov ntshav qabzib ntau ntau.
Ib qho ntxiv, vim li cas cov piam thaj siab ua rau muaj sodium tsawg? Qhov tseeb, qabzib yog ib qho khoom siv osmotic. Yog li, nyob rau hauv cov ntaub ntawv ntawm cov cim hyperglycemia Posm yog nce ua rau kev txav ntawm cov dej tawm ntawm cov hlwb thiab tom qab ntawd txo cov ntshav. sodium qib (dilutional hyponatremia)
Ua li no, vim li cas hyperglycemia ua rau hypernatremia?
Feem ntau ua ntawm hypernatremia vim osmotic diuresis hyperglycemia hauv cov neeg mob nrog ntshav qab zib . Vim yog qabzib ua tsis nkag mus rau cov cell thaum tsis muaj insulin, hyperglycemia ntxiv dehydrates ICF compartment.
Cov electrolyte twg cuam tshuam ntau tshaj los ntawm hyperglycemia?
Lub sijhawm no ob leeg hyperglycemia thiab hyperosmolarity tsav cov dej hloov pauv uas ua rau lub cev qhuav dej thiab poob ntawm electrolytes . Ob tug feem ntau tseem ceeb electrolytes depleted yog sodium thiab potassium. Lawv tshem tawm sai dua los ntawm kev nce osmotic diuresis, lossis tso zis ntau dhau.
Pom zoo:
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Dab tsi yog qhov ua rau hyperglycemia? Hla los yog hnov qab koj cov tshuaj insulin lossis tshuaj txo cov piam thaj hauv qhov ncauj. Noj zaub mov tsis raug. Noj zaub mov ntau dhau. Kab mob. Ua mob. Ua kom muaj kev nyuaj siab. Kev ua haujlwm tsawg
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Hyperglycemia hais txog qib qab zib, lossis piam thaj hauv cov ntshav. Nws tshwm sim thaum lub cev tsis tsim lossis siv cov tshuaj insulin txaus, uas yog cov tshuaj hormones uas nqus cov piam thaj rau hauv cov cell rau siv ua lub zog. Ntshav qab zib hauv ntshav yog qhov qhia ua rau ntshav qab zib
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Hyperglycemia tuaj yeem ua rau hyperkalemia hauv cov neeg mob ntshav qab zib nrog insulin tsis txaus [1,2]. Txawm li cas los xij, hauv cov tib neeg noj qab haus huv, mob hyperglycemia zoo li txo qis lossis tsis cuam tshuam rau cov ntshav hauv cov ntshav potassium [3] thiab hyperkalemia tsis tshua tshwm sim vim lub raum lub peev xwm los tso cov poov tshuaj
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Nov yog cov laj thawj vim li cas cov cell faib. Vim li cas 1: Mitosis thiab meiosis yog cov txheej txheem tseem ceeb. Vim li cas 2: Cell faib rau lub cev kev loj hlob thiab kev loj hlob. Yog vim li cas 3: Cov cell faib los kho peb lub cev. Kev faib tawm ntawm tes yog qhov tseem ceeb rau peb lub cev kev loj hlob thiab kev loj hlob
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Hyperglycemia ua rau cov ntaub so ntswg puas tsuaj los ntawm tsib lub tswv yim tseem ceeb: Kev nce ntxiv ntawm cov piam thaj thiab lwm cov suab thaj los ntawm txoj hauv kev polyol, nce intracellular tsim ntawm qib siab glycation kawg-cov khoom (AGEs), nce kev qhia ntawm receptor rau qib siab glycation kawg cov khoom thiab nws cov ligands activating