![Dab tsi yog pathophysiology ntawm mob plab? Dab tsi yog pathophysiology ntawm mob plab?](https://i.answers-medical.com/preview/medical-health/14141156-what-is-the-pathophysiology-of-abdominal-pain-j.webp)
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2024 Tus sau: Michael Samuels | [email protected]. Kawg hloov kho: 2023-12-16 01:44
Pathophysiology . Visceral mob los ntawm plab viscera, uas yog sab hauv los ntawm cov leeg nqaij uas tsis muaj zog thiab teb feem ntau rau qhov kev xav ntawm qhov tsis sib xws thiab cov leeg sib zog-tsis txiav, kua muag, lossis ua rau khaus hauv zos. Cov txheej txheem ua ntej (lub plab, duodenum, daim siab, thiab txiav txiav) ua rau sab saud mob plab
Hauv qhov no, dab tsi yog pathogenesis ntawm mob plab?
Mob plab tuaj yeem raug ntxias los ntawm stimulation ntawm visceral nociceptors. Kev ua kom cov nociceptors feem ntau xav tau kev nkag siab yav dhau los los ntawm cov xwm txheej pathological, xws li mob, ischemia lossis acidosis. Ob qho xwm txheej ua rau muaj kev sib kis mob mus rau lwm thaj tsam ntawm lub cev thiab kev nthuav dav ntawm mob kev xaav.
Ib yam li ntawd, qhov tshwm sim ntawm qhov mob plab yog dab tsi? Qhia tawm ntawm Pinterest xeev siab, ntuav, thiab quav quav tuaj yeem nrog mob plab yog tias qhov ua rau mob plab hnyuv. Cov teeb meem digestive yog suav tias yog feem ntau ua rau mob plab . Tsis xis nyob lossis qhov tsis xwm yeem hauv ib qho khoom nruab nrog lossis ib feem ntawm plab tuaj yeem ua rau mob uas radiates thoob plaws hauv cheeb tsam.
Zoo sib xws, dab tsi ua rau mob plab hnyav?
Qee qhov feem ntau ua rau ntawm mob plab Cov kab mob no yog kab mob appendicitis, kab mob gastroesophageal reflux (GERD), pancreatitis, kab mob gallbladder, diverticulitis, thiab cov plab hnyuv me.
Cov tsos mob thiab cov tsos mob ntawm tus mob plab yog dab tsi?
Cov tsos mob feem ntau suav nrog mob plab heev mob . Ua npaws, cem quav, tso quav tso quav, rov ua rau lub plab, plab nruj thiab tiv thaiv, tsam plab, jaundice, colic, thiab tsiv chaw mob kuj yuav tshwm sim.
Pom zoo:
Dab tsi yog pathophysiology ntawm mob hnyav?
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Kev mob hnyav yog ib qho qauv dav dav ntawm kev tiv thaiv kab mob rau Cell raug mob tshwj xeeb los ntawm kev nce sai ntawm cov cell tiv thaiv ntawm qhov chaw raug mob. Cov lus teb mob hnyav tau pib los ntawm ob lub cev tiv thaiv kab mob thiab parenchymal ntawm qhov chaw raug mob thiab tau koom tes los ntawm ntau yam kev daws teeb meem nruab nrab
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Qhov tseem ceeb AKI yog lo lus siv los piav qhia txog kev mob tshwm sim uas tshwm sim thaum lub raum ua haujlwm tau poob qis mus rau qhov uas lub cev khaws cov khoom pov tseg thiab dhau los ua tsis muaj peev xwm tswj tau cov roj ntsha, kua qaub-puag thiab dej sib npaug. Cov kab mob pathophysiology ntawm AKI yog ntau yam ua haujlwm thiab nyuaj
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Pathophysiology. Hauv kev yooj yim mechanical obstruction, blockage tshwm sim yam tsis muaj vascular cuam tshuam. Kev noj cov kua dej thiab zaub mov, digestive secretions, thiab gas accumulates saum lub qhov thaiv. Strangulating obstruction yog kev cuam tshuam nrog cov ntshav txaus; Nws tshwm sim nyob rau hauv ze li ntawm 25% ntawm cov neeg mob uas muaj cov kab mob me me
Dab tsi yog qhov txawv nruab nrab ntawm lub plab plab thiab lub plab zom mov?
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Cov plab hauv plab yog qhov tsis txaus ntseeg hauv lub plab uas qhia txog kev nkag mus rau 3-5 tubular zoo li lub plab hnyuv. Lawv tob hauv cov pylorus dua li lawv nyob hauv lwm qhov ntawm lub plab. Tib neeg lub plab muaj ob peb plhom ntawm cov qhov no uas teev rau saum npoo ntawm daim tawv nqaij epithelium
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Feem ntau tus kab mob no kis los ntawm kev mus tom cov tsiaj txhu uas ua rau kis tus kab mob sib kis nrog lawv cov qaub ncaug. Tus kab mob no tsis tuaj yeem nkag rau ntawm daim tawv nqaij. Tom qab nkag mus, tus kab mob khi rau ntawm tes receptors. Cov kab mob tuaj yeem rov ua dua nyob rau hauv cov nqaij ntshiv ntawm cov leeg nqaij ore ncaj qha cuam tshuam rau cov leeg hlwb