![Dab tsi ua rau gingival o? Dab tsi ua rau gingival o?](https://i.answers-medical.com/preview/medical-health/13984748-what-causes-gingival-enlargement-j.webp)
Video: Dab tsi ua rau gingival o?
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2024 Tus sau: Michael Samuels | [email protected]. Kawg hloov kho: 2023-12-16 01:44
1. Cov pos hniav mob kev loj hlob . Gingival hyperplasia tuaj yeem tshwm sim raws li qhov tshwm sim ncaj qha ntawm qhov mob. Qhov mob yog feem ntau ua los ntawm cov plaque buildup ntawm cov hniav los ntawm cov zaub mov, kab mob, thiab kev tu cev tsis zoo.
Ua li no, yam tshuaj twg ua rau cov hniav loj tuaj?
Drug-induced gingival overgrowth yog ib qho kev mob tshwm sim cuam tshuam nrog 3 hom tshuaj: anticonvulsant ( phenytoin (immunosuppressant), cyclosporine A), thiab ntau yam calcium channel blockers ( nifedipine , verapamil , diltiazem).
Tsis tas li ntawd, vim li cas kuv cov pos hniav thiaj loj tuaj? Gingivitis. Gingivitis yog feem ntau ua rau cov pos hniav o . Nws yog ib cov pos hniav kab mob uas ua rau koj cov pos hniav los ua kev chim thiab o . Gingivitis feem ntau yog tshwm sim los ntawm kev tu lub qhov ncauj tsis zoo, uas tso cai rau cov quav hniav los tsim cov kab mob. cov pos hniav kab thiab cov hniav
Ua qhov no hauv kev txiav txim siab, qhov kev loj hlob ntawm cov hniav cuav yog dab tsi?
Gingival o loj yog qhov nce ntawm qhov loj ntawm cov hniav ( cov pos hniav ). Nws yog ib qho kev nthuav dav ntawm gingival kab mob. Gingival o tuaj yeem tshwm sim los ntawm ntau yam, nrog rau cov mob inflammatory thiab cov kev mob tshwm sim ntawm qee yam tshuaj. Kev kho mob yog nyob ntawm qhov ua rau.
Puas yog gingival hyperplasia thim rov qab?
Gingival hyperplasia yog qhov tshwm sim tsis zoo ntawm qee yam tshuaj (phenytoin, calcium antagonists thiab cyclosporine). Yeeb tshuaj gingival hyperplasia kev kho mob yog thim rov qab thaum qhov kev kho mob raug tso tseg lossis hloov kho. Yog tias kev hloov tshuaj tsis tuaj yeem ua rau lub qhov ncauj huv thiab tiv thaiv kab mob yog qhov tseem ceeb.
Pom zoo:
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Gingival overgrowth (GO) yog cov kev mob tshwm sim cuam tshuam nrog qee cov chav kawm sib txawv ntawm cov tshuaj, xws li anticonvulsants, tiv thaiv kab mob, thiab calcium channel blockers. Ib qho ntawm cov tshuaj tseem ceeb cuam tshuam nrog GO yog antiepileptic phenytoin, uas cuam tshuam rau cov ntaub so ntswg sib txuas los ntawm kev hloov pauv cov txheej txheem hloov pauv sab nraud
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Zoo li feem ntau qhov kev kho mob ib txwm muaj txiaj ntsig thiab ntau yam, lub ntsiab lus tuaj yeem sib txawv me ntsis, tab sis feem ntau cov ntsiab lus hauv qab no siv: Ua rau me me hypoxemia: PaO2 = 60 txog 79 mmHg. Qhov nruab nrab hypoxemia: PaO2 = 40 txog 59 mmHg. Hypoxemia hnyav: PaO2 <40 mmHg
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Qhov feem ntau raug zam txim thiab raug tshuaj ntsuam xyuas thiab ntsuas cawv yog: 5-panel panel drugurine test, uas txheeb xyuas cov zis zis rau cov yeeb tshuaj xas, cocaine, PCP, amphetamines, thiab opiates. Ntau qhov kev sim tshuaj ntsuam xyuas kev sim tshuaj kuj tseem ntxiv cawv rau 5-paneltest
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Ntawm cov tshuaj tiv thaiv hypertensives, nifedipine tau pom ntau dua ntawm cov gingival o. [4] Nws pib raws li qhov loj ntawm interdental papilla thiab tom qab ntawd hloov mus rau hauv ib qho lobulated loj lossis nodules. Nws kuj tseem tuaj yeem ncav cuag lub crown thiab ncav cuag qhov siab occlusal, yog li cuam tshuam nrog mastication