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Video: Dab tsi yog pathophysiology ntawm bronchitis?
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2024 Tus sau: Michael Samuels | [email protected]. Kawg hloov kho: 2023-12-16 01:44
Thaum lub sij hawm ib qho kev mob mob ntsws , cell cov bronchial - cov ntaub so ntswg yog irritated thiab mucous daim nyias nyias ua hyperemic thiab edematous, diminishing bronchial mucociliary muaj nuj nqi. Hauv kev teb, copious secretion ntawm mucus tsim, uas ua rau tus yam ntxwv hnoos ntawm mob ntsws.
Hauv qhov no, dab tsi yog pathology ntawm mob ntsws ntev?
Cov pathology ntawm mob ntsws ntev muaj xws li ib tug inflammatory mononuclear cell infiltrate nyob rau hauv lub pa phab ntsa thiab ib tug neutrophil influx mus rau hauv lub pa lumen. Cov txheej txheem molecular uas ua rau mob thiab nws lub luag haujlwm pathogenetic hauv kev ua rau cov hnoos qeev ua haujlwm tau pib piav qhia.
Tom qab ntawd, lo lus nug yog, dab tsi yog qhov tshwm sim ntawm kev mob ntsws ntev? Mob bronchitis yog qhov kev txhim kho sai sai ntawm kev mob hauv cov hlab ntsws-txoj hlab pa loj rau hauv koj lub ntsws. Nws feem ntau tshwm sim vim muaj tus kab mob los yog ua pa ntawm tej yam uas ua rau lub ntsws ua pa xws li haus luam yeeb, pa taws, plua plav thiab huab cua. Kab mob qee zaum ua rau mob ntsws ntev.
Ib yam li ntawd, nws tau nug, dab tsi yog txheej txheem mob ntsws?
Mob mob ntsws feem ntau tshwm sim peb mus rau plaub hnub tom qab mob khaub thuas lossis mob khaub thuas. Nws yuav pib nrog hnoos qhuav, tom qab ob peb hnub hnoos hnoos tuaj yeem ua rau hnoos qeev. Cov neeg feem ntau tau txais qhov kev sib tw hnyav ntawm mob ntsws hauv ob rau peb lub lis piam, txawm hais tias hnoos qee zaum tuaj yeem ua rau plaub lub lis piam lossis ntau dua.
Dab tsi yog pathophysiology ntawm mob ntsws?
Feem ntau mob ntsws o tshwm sim thaum muaj kev puas tsuaj hauv koj lub cev tiv thaiv ntuj tso cai rau cov kab mob nkag mus thiab sib kis hauv koj lub ntsws. Txhawm rau rhuav tshem cov kab mob tawm tsam, cov qe ntshav dawb sai sai. Nrog rau cov kab mob thiab cov fungi, lawv sau cov hnab pa hauv koj lub ntsws (alveoli). Ua pa tuaj yeem ua haujlwm.
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Hom 1 mob ntshav qab zib tshwm sim thaum qee qhov lossis tag nrho cov cell tsim cov tshuaj insulin hauv cov txiav txiav. Qhov no txhais tau tias nws pib thaum lub cev tiv thaiv kab mob tawm tsam cov cell hauv lub cev. Hauv hom 1 mob ntshav qab zib, lub cev tiv thaiv kab mob tsim cov tshuaj insulin (beta cells) hauv cov txiav txiav
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Kev mob hnyav yog ib qho qauv dav dav ntawm kev tiv thaiv kab mob rau Cell raug mob tshwj xeeb los ntawm kev nce sai ntawm cov cell tiv thaiv ntawm qhov chaw raug mob. Cov lus teb mob hnyav tau pib los ntawm ob lub cev tiv thaiv kab mob thiab parenchymal ntawm qhov chaw raug mob thiab tau koom tes los ntawm ntau yam kev daws teeb meem nruab nrab
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Qhov tseem ceeb AKI yog lo lus siv los piav qhia txog kev mob tshwm sim uas tshwm sim thaum lub raum ua haujlwm tau poob qis mus rau qhov uas lub cev khaws cov khoom pov tseg thiab dhau los ua tsis muaj peev xwm tswj tau cov roj ntsha, kua qaub-puag thiab dej sib npaug. Cov kab mob pathophysiology ntawm AKI yog ntau yam ua haujlwm thiab nyuaj
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Pathophysiology. Hauv kev yooj yim mechanical obstruction, blockage tshwm sim yam tsis muaj vascular cuam tshuam. Kev noj cov kua dej thiab zaub mov, digestive secretions, thiab gas accumulates saum lub qhov thaiv. Strangulating obstruction yog kev cuam tshuam nrog cov ntshav txaus; Nws tshwm sim nyob rau hauv ze li ntawm 25% ntawm cov neeg mob uas muaj cov kab mob me me
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Mob glomerulonephritis (GN) muaj xws li cov kab mob hauv lub raum uas ib qho kev tiv thaiv kab mob ua rau mob thiab loj hlob ntawm cov ntaub so ntswg glomerular uas tuaj yeem ua rau muaj kev puas tsuaj rau hauv qab daus daim nyias nyias, mesangium, lossis capillary endothelium